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All of the Modules in the Mastering the Gut are below - as you finish each lesson, the next one will become available!

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Introduction to Functional Splanchnology

Introduction to Functional Splanchnology

Introduction to analysis and rehabilitation of gastrointestinal dysfunction using Neuro-Endo-Immune Supersystem-based organ rehabilitation that assesses the visceral conditions of a patient with reoccurring viscerosomatic referred pain, subluxation and degenerative disc disease.

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Tenets of Functional Splanchnology

A tenet serves as part of a primary basis of an ideology, and it cannot be changed or discarded without affecting the very system's paradigm. An example of a tenet of Functional Splanchnology is: "No part of the body can be understood separate from the whole."

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Correlation to Chiropractic

The traditional chiropractic model for organ dysfunction is a vertebral subluxation with cord level irritability firing to the sympathetic nerves. This causes a hypersympathetic response but due to philosophy based anatomy, physiology and neurology. There is no mention of what occurs with the blood supply to the organ. During a hypersympathetic response, the blood vessels are vasocontricted reducing the blood supply necessary for proper organ function.

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Altered Structure - Altered Function

Structure always affects function. Function always affects structure. This tenet can be applied to every part and tissue of the body. How does altered structure affect gastrointestinal function?

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Bridging the Lab Gap

All nerves depend wholly upon the arterial system for their nutrition and the quality of their function, such as sensation, signal transmission and motion, even though by the law of reciprocity they furnish force, nutrition and sensation to the artery itself.

  • Vasomotor nerves control the diameter of the blood vessels.
  • Nerves do not control what is flowing through the blood vessels.

Red Blood Cell Morphology

Vasoconstriction, ischemia, red blood cell morphology and anemia cause decreased function at the cellular level in any body tissue where it is occurring.

Unit 1

Red Blood Cell Morphology

Anemia is functionally defined as an insufficient red blood cell mass to adequately deliver oxygen to peripheral tissues. However, this assumes the cells are of normal shape and size. This also assumes if there is a problem, it can involve only one of the specific diagnosable anemia’s.

Unit 2

Macrophage Response

Macrophages (and their precursors, monocytes) are the 'big eaters' of the immune system. These cells reside in every tissue of the body, albeit in different guises — such as microglia, Kupffer cells and osteoclasts.

Unit 3

Red Blood Cell Morphology Practical Application

During their 120-day life span, human RBCs repeatedly traverse capillaries of the vascular bed and interendothelial slits of the venous sinus of spleen red pulp. This necessitates maintenance of the ability of RBCs to undergo repeated, extensive, and reversible deformations. Repeated major membrane deformations induce changes in the RBCs size and shape.

Unit 4

Download: Red Blood Cell Morphology Analysis Guide

Red Blood Cell Morphology Practical Application

This module bridges the lab gap using Red Blood Cell indices to identify possible morphologic changes in red blood cells contributing to the patient's signs and symptoms.

Unit 5

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Splanchnology

The health of the body is dependent upon the health of the nerves and the blood supply. The health of the nerves is dependent upon the blood supply for normal spinal cord and nerve cell function.

Gastrointestinal Barrier Variables

Impaired Gastrointestinal Barrier Variables

Mesentery

The mesentery as exceedingly sensitive to every kind of stimulus, mechanical, chemical and thermal, and advanced the view that true visceral pain primarily arises from the mesentery, partly by impulses arising in the Pacinian corpuscles.

Portal Hypertension

Splanchnoptosis

Gastrointestinal Function Analysis

The health of the gastrointestinal tract is dependent upon the health of the intestinal lining, the barrier variables and the microbial flora. The environment of the gastrointestinal microbes is dependent upon the environment determining their behavior and function.

Gastrointestinal Function Profile Analysis

Gut bacteria influence the gastrointestinal environment and flora. Gut bacteria are involved in multiple organ dysfunction and systemic inflammation.

  • Commensal Bacteria Behavior and Pathogenicity
    • Bacteroidetes Phylum
    • Firmicutes Phylum
    • Actinobacteria Phylum
    • Proteobacteria Phylum
    • Eurarchaeota Phylum
    • Fusobacteria Phylum
    • Verrucomicrobia Phylum
  • Opportunistic Bacteria Behavior and Pathogenicity
  • Pathogenic Bacteria Behavior and Pathogenicity
  • Mold/Yeast/Fungi Behavior and Pathogenicity
    • PCR vs. Culture
  • Parasite Behavior and Pathogenicity
    • Helminth Phylum
    • Protozoa Phylum
    • PCR vs. Optimized Parasite Recovery
  • Digestion and Absorption
    • Products of Protein Breakdown (Valerate, Isobutyrate, Isovalerate)
    • Fecal Fat
    • Long-Chain Fatty Acids
    • Cholesterol
    • Phospholipids
  • Inflammation and Immunology
    • Calprotectin
    • Eosinophil Protein X
    • Fecal Secretory IgA
  • Gastrointestinal Microbiome
    • Short-Chain Fatty Acids Total
    • n-Butyrate concentrations
    • n-Butyrate %
    • Acetate %
    • Propionate %
    • Beta-glucuronidase
  • pH and Zeta Potential
  • DRG labs GI Pathogens Plus Panel
  • Genova 2200 Gastrointestinal Function Profile Analysis

Oral Bacteria

Oral bacteria influence the gastrointestinal environment and flora. Oral bacteria are involved in thyroid dysfunction.

  • Oral Health
  • Aa-Complex
  • Red Complex
  • Orange Complex
  • Orange-Associated Complex
  • Green Complex
  • Systemic Diseases associated with Oral Infections